BDNF and cognition: These deficits were not associated with changes in hippocampal LTP, suggesting that mechanisms other than hippocampal synaptic plasticity as modeled by LTP were involved in the regulation of mnesic performances, or that the chromatin remodeling leading to BDNF signaling upregulation could reverse some synaptic plasticity dysregulations but were not enough to counteract all the cognitive deficits induced by ethanol.