Since Ang‐2 is regulated by oxygen levels, we also investigated the surrogate hypoxia markers carbonic anhydrase IX (CAIX) in human GBM (Fig 6G and H) and glucose transporter‐1 (GLUT1) in murine GBM (described in Fig 4F, see above), and compared those findings to the size of gross hemorrhagic and necrotic areas in treated versus treatment‐naive GBM. Here, SLC2A1 is linked to glioblastoma.