FASN and viral infectious disease: We hypothesize that percent cell viability for drug-treated PIV3- or HRV-infected cells appears to be higher than for drug-treated mock-infected cells because drug treatment may protect the infected cells from the cytopathic effect of these viruses by reducing the viral load, or viral infection may upregulate cellular lipid metabolic pathways, blunting the effects of FASN inhibition on cell viability.