Mediators of sepsis such as lipopolysaccharide (LPS), a gram-negative bacterial cell wall component, triggers apoptosis in cardiac myocytes by promoting the secretion of cytokines such as tumor necrosis factor α (TNF-α), interleukin-6 (IL-6), IL-10, and interferon (IFN)-γ [3]. This evidence concerns the gene IL6 and Sepsis.