To further examine the impact of the loss of FANCB on stressed hematopoiesis, we injected Fancb−/y mice and their male WT littermates, along with the Fanca−/− mice as a comparative FA model, with the myeloid-ablating agent Fluorouracil (5-FU) and monitored BM recovery over a period of 30 days28. This evidence concerns the gene FANCB and Friedreich ataxia.