AVPR1B and motion sickness: In the present study, we first proved that the bilateral microinjection of AVP into vestibular nuclei, similar to a rotatory stimulus, induced conditioned taste aversion in rats and that the application of SSR149415, an antagonist of AVP V1bR, blunted this CTA-inducing effect of AVP and rotatory stimulus-induced CTA, suggesting that AVP, through its action on the VN, could contribute to the development of motion sickness in rats via the mediation of V1bR.