In other mouse models of SLE, the Ets knockout mouse (Ets−/−) and the New Zealand Black (NZB) × SWR F1 cross (SNF1 mice), increased levels of Th17 cells and IL-17 seem to contribute to the inflammation of kidneys, i.e., glomerulonephritis (64, 65). The gene discussed is IL17A; the disease is systemic lupus erythematosus.