According to Steinke et al. [93], high levels of mentioned IFN-γ distinguish AERD (aspirin-exacerbated respiratory disease) from aspirin tolerant asthma and underlie the robust constitutive and aspirin-induced secretion of CysLTs that characterize this disorder, as AERD is associated with eosinophils maturing locally in a high interferon (IFN)-γ. The gene discussed is IFNG; the disease is asthma.