Statins have previously been described to inhibit ligand-induced EGFR autophosphorylation, to enhance the activity of EGFR-inhibitor gefitinib in different cancer cell lines and primary tumor cultures, and improve progression free survival in NSCLC patients treated with EGFR-tyrosine-kinase-inhibitors [17–20, 22, 23]. The gene discussed is EGFR; the disease is non-small cell lung carcinoma.