Moreover, mutations in neuroligin-3 (Radyushkin et al., 2009), neuroligin-4 (Jamain et al., 2008), or neurexin-2 (Dachtler et al., 2014) produce autism-like phenotypes in mice with changes in the cortical inhibitory network, though not always with deficits in sensory processing which indicate that compensatory mechanisms and brain region specificities should be taken into account. Here, NLGN3 is linked to autism.