On the other hand, the expression of FGF2, as well as CXCR4, CD45 and COL1A1, in the isolated fibrocyte-like cells was similar between the tumours (Fig. 6c), suggesting that the increase of FGF2 in bevacizumab-treated tumours was due to the increase in the number of these cells. This evidence concerns the gene PTPRC and neoplasm.