Moreover, enhanced gene expression for the natural killer cell receptor, NKRP1A, in tumors from mice vaccinated against AMHR2-CD (Figure 5(c)), implies that NK cells may also play a role in the observed tumor immunity perhaps as a result of recruitment through IFNγ-dependent CXCR3 signaling [41] or through an IL-17/CCL2 recruitment mechanism [42]. Here, CXCR3 is linked to neoplasm.