Consistent with these studies, transfection of miR-29b mimic in IPF fibroblasts was found to control the expression of COL1, in both TGF-β1 treated as well as baseline conditions (Montgomery et al., 2014), further underscoring the anti-fibrotic potential of miR-29 in lung fibroblasts. This evidence concerns the gene TGFB1 and idiopathic pulmonary fibrosis.