In this context, the antioxidant and anti-inflammatory natural compound resveratrol with beneficial properties on mitochondrial energy metabolism [125] and FAO [126] was shown to improve mitochondrial FAO capacities in fibroblasts from human VLCAD- and carnitine palmitoyltransferase (CPT)2-deficient patients by increasing the expression of VLCAD and CPT2 proteins. This evidence concerns the gene CPT2 and very long chain acyl-CoA dehydrogenase deficiency.