Influenza A and B viruses evolve at high rates with mutations being prevalent in their hemagglutinin genes encoding glycoprotein (HA), the major target for neutralizing antibodies.1 Amino acid substitutions in antibody‐binding sites cause antigenic drift, which, in its turn, results in the occurrence of seasonal influenza epidemics of different severity.2, 3 To overcome antigenic drift, continuous virologic and disease surveillance is undertaken to ensure that viruses included in seasonal influenza vaccines and circulating viruses are antigenically similar. Here, ART4 is linked to influenza.