Clonal Ae. albopictus C6/36 cells stably expressing the trans-splicing deficient ΔCHIKV/DENVv1-ΔN Bax, ΔDENV/CHIKVv1-ΔN Bax, ΔCHIKV/DENVv1-ΔN Bax, or ΔDENV/CHIKVv1-ΔN Bax constructs did not display observable DNA fragmentation upon infection with CHIKV (Fig 7A) or DENV (Fig 7B and S1 Fig through S4 Fig), further demonstrating that the insertion of a UAA codon in the P9.0 helix of the group I intron prevents premature expression of the ΔN Bax effector gene. This evidence concerns the gene BAX and infection.