GAD1 and schizophrenia: Prenatal lead exposure results in a loss of parvalbumin-interneurons co-labeled with GAD67 protein in specific brain regions such as medial prefrontal cortex, striatum, and hippocampus but to increased activity of the subcortical dopaminergic system and intensified locomotor response to cocaine in a rat model of schizophrenia (Stansfield et al., 2015).