Alterations in calretinin expression have been observed in mouse models of epilepsy and psychiatric diseases (Shin et al., 2013); however, no significant differences in the number of calretinin-immunoreactive interneurons in the cerebral cortices of wild-type and DBZ (DISC1-binding zinc finger protein) knockout (KO) mice have been reported (Koyama et al., 2013). This evidence concerns the gene ZNF365 and psychiatric disorder.