MKI67 and neoplasm: No significant difference in CXCR4 genotype distribution was observed according to clinicopathological features analyzed such as tumor histology (p = 0.686), nuclear grade (p = 0.312), nodal status (p = 0.697), estrogen receptor status (p = 0.630), progesterone receptor status (p = 0.287), p53 (p = 0.789), Ki67 (p = 0.129), and HER-2 status (p = 0.818) (Table 2).