We found that BaP or CXCL13 treatment or SPP1 overexpression in lung epithelial or cancer cells did not up-regulate RANKL or Src (data not shown), but did activate β–catenin (Figure 6—figure supplement 1), suggesting that CXCL13 may induce an EMT via different mechanisms in different settings. The gene discussed is CXCL13; the disease is cancer.