GAD1 and aceruloplasminemia: In the 1980s, some of these cases were reported in association with autoantibodies, and three clinical entities have been established since then, based on the type of the associated antibodies (Abs) and specific clinical features: gluten ataxia (GA), anti-glutamic acid decarboxylase antibodies (GAD Abs)-associated cerebellar ataxia (GAD Abs-CA), and Hashimoto’s encephalopathy (HE).