NAC has been shown to inhibit epithelial–mesenchymal transition (EMT) in rat alveolar epithelial cells (50), to diminish TGF-β-induced gel contraction, fibronectin (Fn), and VEGF production as well as α-SMA expression in human lung fibroblasts (51) as well as the concentrations of several cytokines produced by alveolar macrophages of IPF patients (52, 53). Here, FN1 is linked to idiopathic pulmonary fibrosis.