Lee et al. reported that IL-17 increased the expression of Toll-like receptor 3 via the STAT3 pathway in rheumatoid arthritis (RA) fibroblast-like synoviocytes and that STAT3 phosphorylation was increased in the synovial samples from patients with RA compared with patients with OA [39]. The gene discussed is STAT3; the disease is rheumatoid arthritis.