In mice pretreated with anti-mouse P-selectin antibody, MM cells displayed delayed extravasation with approximately 2.7-fold more MM cells still present in the circulation, whereas, in mice pretreated with anti-mouse PSGL-1 antibody and anti-human SelK2, there were 1.4-fold more MM cells in the circulation compared to untreated mice at time 50 minutes (Figure 2(a)). The gene discussed is SELPLG; the disease is Miyoshi myopathy.