This is in agreement with our results showing that the expression of GnT-IVa substantially increased in HCC tissues and the products of the GnT-Iva-catalyzed, core-α-1, 6-fucosylated triantennary glycan increased on the cell surface and in the serum of the same HCC patients. The gene discussed is MGAT4A; the disease is hepatocellular carcinoma.