Figure1 compares the steady-state current density at −80 mV of ENaC containing PHA1-causing mutations, including the S562P missense mutation in αENaC and the K106_S108delinsN in γENaC; and the N530S missense mutation in γENaC associated with Liddle’s Syndrome with that of wild-type ENaC expressed in CHO cells. This evidence concerns the gene SCNN1G and Liddle syndrome.