ART4 and viral infectious disease: To exclude that elimination of T-AgNP expressing cells was due to some unspecific effects of inflammation during viral infection rather than to a specific immune response against the immune-dominant LCMV epitope NP118–126, additional groups of mice were infected with VV recombinants encoding either the LCMV glycoprotein precursor (VV-G2) or the LCMV nucleoprotein (VV-YN4) (Fig. 4C) [13].