Fortunately, because of the highly favorable interleukin (IL)-28B genotype (CC genotype rs12979860 in 75.1–84.1%) [16, 17], the reported SVR rate in patients with chronic hepatitis C in Asia treated with PEG-IFN/RBV regimens (61–79%) is higher than that in Caucasians receiving PEG-IFN/RBV or triple regimens containing HCV protease inhibitors (38–41%) [18–21]. Here, IFNA1 is linked to chronic hepatitis C virus infection.