Efficient correctors also need to be developed for other ABC processing mutants that cause disease such as ABCG2 (gout), ABCB4/ABCB11 (progressive familial intrahepatic cholestasis), ABCA1 (Tangier disease), ABCC2 (Dubin-Johnson syndrome), ABCC6 (pseudoxanthoma elasticum), and ABCC8 (hyperinsulinimic hypoglycemia of infancy). This evidence concerns the gene ABCC8 and Dubin-Johnson syndrome.