CXCL10 and myocardial infarction: Inducible expression of IP10 has been demonstrated in monocytes, endothelial cells and fibroblasts [42,43] and regulates the reparative response in an experimental model of myocardial infarction [44] To our knowledge, no previous study reported on the functional effects of CXCR3 agonists on the response of the heart to hemodynamic overload.