IGF1R and neuroendocrine neoplasm: As IGF1-R dependent signaling has been shown to promote tumorigenesis and hypersecretion syndromes in neuroendocrine tumors of the gastroenteropancreatic system (GEP) [20–22], we investigated these IGF1-R-targeting immunoliposomes in a preclinical xenograft model with promising therapeutic potential for the treatment of GEP-NETs.