EBV-miR-BART10-3p could promote invasion and migration of NPC cells, through a mechanism underlying the inhibition of BTRC expression, thereby abolishing the activity of βTrCP E3 ubiquitin ligase, leading to increased β-catenin and Snail that are two substrates of βTrCP and important roles in EMT. This evidence concerns the gene SNAI1 and nasopharyngeal carcinoma.