The first two pathways that are worth mentioning are associated (i) with the ErbB receptor signaling and (ii) with genes (c-KIT, STAT3, AKT3) that have been mainly described as increased in acute myeloid leukemia, in which the crucial role of CEBPA has been widely demonstrated [26, 27]. This evidence concerns the gene AKT3 and acute myeloid leukemia.