Alzheimer’s disease (AD) is one of the major amyloidoses, with two types of amyloid deposited in the brain: (i) amyloid β-peptide (Aβ) forming aggregates senile plaques and cerebrovascular amyloid angiopathy [1]; and (ii) tau protein, which forms neurofibrillary tangles, neuropil threads, and dystrophic neurites [2]. This evidence concerns the gene MAPT and Alzheimer disease.