And the present data demonstrate that: 1) SX can obviously reduce the levels of cardiac enzyme CK-MB, LDH and MDA and increase the activity of SOD; 2) SX can markedly reduce the myocardial infarct size, the degree (∑-ST) and scope (N-ST) of myocardial ischemia; 3) SX can reduce MBP, LVSP, CVR and increased CBF, MBF; 4) SX can significantly reduce myocardial oxygen extraction ratio. The gene discussed is SOD1; the disease is myocardial infarction.