Yet, in the inflammation process, the resolution phase is multifaceted38; in addition to the up-regulation of the genes, which is a hallmark of the AA phenotype, such as Arg1, and necessary for the diminution of the activity of the inflammatory molecules produced, other cytokines (e.g., IL10) are generated to repair the damaged tissues in the AD phenotype. The gene discussed is IL10; the disease is Alzheimer disease.