CP and Wilson disease: They allow us to suggest that at this time in the liver (i) mechanisms of copper accumulation and its redistribution in organelles parallels those in patients with Wilson disease; (ii) in newborns, CTR1 is neither a major importer of copper no a copper donor for Cu(I)-chaperons; (iii) gene expression profile of a copper transporting proteins is adapted to the main nutrition source of copper, i. e. milk Cp.