MCL1 and acute lymphoblastic leukemia: Transient MCL-1 knockdown by siRNA, or the MCL-1 inhibitor Obatoclax sensitized SupB15 and SupB15MR cells to H2O2 cytotoxicity (Figures 6A, 6B, and Supplementary Figure S5), suggesting that MCL-1 contributes to the redox adaptation process in ALL.