Previous studies have already categorized CCNJ as a putative oncogene: it was found to be embedded in a cluster of dysregulated genes in pediatric high-risk B-precursor acute lymphoblastic leukemia;31 report from Feliciano et al.32 showed that it was not expressed in normal HMEC cells, but in breast cancer cells MCF7 and MDA-MB-231, and when CCNJ was silenced, the proliferation of MCF7 cells were decreased due to G2/M cell cycle arrest. Here, CCNJ is linked to breast carcinoma.