To further explore this dichotomy of blood TH1/TH2 equilibrium, Nureki and colleagues [59] showed that under unstimulated conditions, TH1 and TH2 chemokines (interferon-inducible protein-10 (IP-10) and thymus and activation-regulated chemokine (TARC)) were both increased in the serum of sarcoidosis patients. This evidence concerns the gene CCL17 and sarcoidosis.