Huber et al. demonstrated that parathyroid hormone serves as a DPP4 inhibitor and increases cardiac SDF-1 level, which in turn enhances CXCR4+ bone marrow-derived stem cell homing to ischemic heart and attenuates ischemic cardiomyopathy after infarction (91). Here, CXCL12 is linked to ischemic cardiomyopathy.