SMARCA4 and neoplasm: Whereas mutations in the SMARCA4 gene and certain similarities to malignant rhabdoid tumors are known for this cancerous disease [14–16, 20] further characterization of the corresponding cellular models SCCOHT-1 and BIN-67 demonstrated significant differences in surface marker and filament expression compared to ovarian adenocarcinoma cells and therefore confirmed SCCOHT as a separate tumor entity with distinct growth properties.