When lymph nodes were analyzed on day 7 post-infection, we found that Hector T cells differentiated into IL-17A-secreting effector cells in response to C. albicans in B6 mice, but their expansion and differentiation was strongly reduced in CCR7-deficient mice (Fig 3F), indicating that the delivery of C. albicans-derived antigen to the cervical lymph nodes was cell-associated and dependent on CCR7-mediated cell trafficking in vivo. The gene discussed is CCR7; the disease is infection.