ZNF217 and breast neoplasm: Altogether, clinical strategies counteracting ZNF217-mediated effects, either directly (miRNA or siRNA ZNF217 silencing) or by targeting its possible key-mediators (Aurora-A, FAK, PI3K/Akt and TGF-β signaling pathways) would represent valuable alternative approaches for the management of the subpopulation of ZNF217+ breast tumors possessing poor prognosis.