Genetic analysis in patients with Brugada syndrome has shown an association with sodium channel SCN5A mutations in about 20% of the cases.9 This mutation results in an accelerated inactivation of sodium channels, which predisposes them to ventricular arrhythmias.1 Our patient had Brugada Type 1 ECG pattern induced by fever. The gene discussed is SCN5A; the disease is Ventricular arrhythmia.