Furthermore, Dinulescu et al. forced expression of oncogenic KRAS or conditional PTEN deletion in ovarian surface epithelium of a mouse model obtaining preneoplastic ovarian lesions with an endometrial glandular morphology, while the combination of both gave rise to invasive and widely metastatic endometrioid ovarian cancer [72]. The gene discussed is KRAS; the disease is ovarian endometrioid adenocarcinoma.