Some conventional chemotherapeutics (e.g., the nucleoside analog gemcitabine) as well as targeted agents [e.g., the epidermal growth factor receptor (EGFR) inhibitor, erlotinib] result in increased levels of class I or class II MHC molecules on the tumor cell surface, effectively facilitating their recognition by the immune system [6, 7]. Here, EGFR is linked to neoplasm.