BCR and neoplasm: 2008). The introduction of miR-203 inhibited the expression of c-Abl and BCR-c-Abl in tumor cells, and attenuated tumor cell proliferation in c-Abl-dependent manner. Using an c-Abl mRNA 3′ UTR reporter, Bueno et al. demonstrated that miR-203 directly binds to the 3′ UTR of c-Abl mRNA (Bueno et al. 2008; Craig et al. 2011).