In previous study, mannosyl (alpha-1,6-)-glycoprotein β -1,6-N-acetyl-glucosaminyltransferase (MGAT5) and Core 2 β -1, 6-N-acetylglucosaminyltransferase-1 (GCNT1) formed GlcNAc β1,6 branching glycan increased PCa aggressiveness [18, 19]. The gene discussed is GCNT1; the disease is posterior cortical atrophy.