Considering our outcomes, as presented in this paper, the GC heterozygotic status at the IL6 −174 G>C SNP, as well as the presence of C alleles at both IL6 and IL1B +3954 C>T SNPs, seems to have been involved in the occurrence of congenital toxoplasmosis in the studied fetuses and neonates. This evidence concerns the gene IL6 and congenital toxoplasmosis.