BCL9 and ductal breast carcinoma in situ: Analysis of the microarray data of the MIND samples showed canonical Wnt signaling to be among the significantly upregulated pathways in our dataset (Fig 1c-d, Additional file 4) and BCL9 was significantly upregulated during the transition from DCIS to IDC (q value <5 %) (Fig. 1c-d and Additional file 3).